Platelet Release and Thromboxane Synthesis

نویسندگان

  • AUKE C. DE BOER
  • ALEXANDER G. G. TURPIE
  • RODNEY W. BuTT
  • RAYMOND V. JOHNSTON
  • EDWARD GENTON
چکیده

out coronary disease. Cathet Cardiovasc Diagn 3: 47, 1977 14. Schnaar RL, Sparks HV: Response of large and small coronary arteries to nitroglycerin, NaNO^, and adenosine. Am J Physiol 22: 223. 1972 15. Harder DR, Belardinelli L, Sperelakis N, Rubio R, Berne RM: Differential effects of adenosine and nitroglycerin on the action potentials of large and small coronary arteries. Circ Res 44: 176, 1979 16. Krishnamurty VSR, Adams HR, Smitherman TC, Templeton GH, Willerson JT: Influence of mannitol on contractile responses of isolated perfused arteries. Am J Physiol 232: H59, 1977 17. Borda L, Schuchleib R, Henry PD: Effects of potassium on isolated canine coronary arteries. Modulation of adrenergic responsiveness and release of norepinephrine. Circ Res 41: 778, 1977 18. Hattenhauer M, Neill WA: The effect of cold air inhalation on angina pectoris and myocardial oxygen supply. Circulation 51: 1053, 1975 19. Pepine CJ, Feldman RL, Conti CR: Action of intracoronary nitroglycerin in refractory coronary artery spasm. Circulation 65: 411 1982

برای دانلود متن کامل این مقاله و بیش از 32 میلیون مقاله دیگر ابتدا ثبت نام کنید

ثبت نام

اگر عضو سایت هستید لطفا وارد حساب کاربری خود شوید

منابع مشابه

Effects of thromboxane A2 inhibition on osteogenic sarcoma cell-induced platelet aggregation.

There is evidence that tumors may stimulate platelet aggregation, causing release of thromboxane A2. Thromboxane A2 may potentiate tumor metastasis by stimulating tumor cell growth and proliferation and by enhancing platelet-tumor cell aggregate formation. Despite potential significance of thromboxane A2 in tumor metastasis, agents which inhibit thromboxane A2 synthesis have not been uniformly ...

متن کامل

Thromboxane synthesis and platelet protein release during simulated extracorporeal circulation.

Platelet secretion induced by certain soluble aggregating agents is associated with thromboxane formation. Activation of platelets by artificial surfaces may involve similar biochemical pathways. Therefore. we monitored platelet release and formation of thromboxane B2. the stable endproduct of thromboxane A2, during simulated extracorporeal circulation where contact between blood and synthetic ...

متن کامل

Role of platelet-prostaglandin synthesis in shear-induced platelet alterations.

Exposure of citrated human platelet-rich plasma (C-PRP) to mechanical shear stress has been shown to induce platelet aggregation (PAG), release from platelets of dense granule contents, and platelet lysis. The mechanisms involved in these shear-induced platelet alterations have not been defined. To examine the possible role of prostaglandin (PG) synthesis in shear-induced platelet reactions, C-...

متن کامل

Fibrinogen mediated activation of platelet aggregation and thromboxane A2 release: pathological implications in vascular disease.

The effect of a human fibrinogen preparation on in vitro platelet aggregation was assessed. Platelets were obtained from healthy volunteers. Human fibrinogen induced platelet aggregation in 65% of platelet rich plasma samples and enhanced submaximal platelet aggregation induced by heparin or by several conventional agonists in all samples. Aggregation induced by fibrinogen alone was reversed by...

متن کامل

A regimen for low-dose aspirin?

The effects of different regimens of 40 mg aspirin on platelet thromboxane A2 synthesis and vascular prostacyclin synthesis were determined in patients who were undergoing elective surgery for removal of varicose veins. Aspirin 40 mg taken at intervals of 48 hours consistently reduced platelet thromboxane A2 synthesis to a level at which it failed to support platelet aggregation and the associa...

متن کامل

Platelet thromboxane release after subarachnoid hemorrhage and surgery.

We studied adenosine diphosphate-induced platelet aggregation and the associated release of thromboxane B2 in platelet-rich plasma from 88 patients with subarachnoid hemorrhage and 26 healthy controls. During the first 3 days after subarachnoid hemorrhage, the patients showed significantly decreased (p less than 0.05) platelet aggregability and thromboxane release relative to the controls, but ...

متن کامل

ذخیره در منابع من


  با ذخیره ی این منبع در منابع من، دسترسی به آن را برای استفاده های بعدی آسان تر کنید

برای دانلود متن کامل این مقاله و بیش از 32 میلیون مقاله دیگر ابتدا ثبت نام کنید

ثبت نام

اگر عضو سایت هستید لطفا وارد حساب کاربری خود شوید

عنوان ژورنال:

دوره   شماره 

صفحات  -

تاریخ انتشار 2005